Milk glucose as an index of the intracellular glucose concentration of rat mammary gland
نویسندگان
چکیده
منابع مشابه
Glucose transporter expression in rat mammary gland.
The expression of different glucose transporter isoforms was measured during the development and differentiation of the rat mammary gland. Before conception, when the mammary gland is mainly composed of adipocytes, Glut 4 and Glut 1 mRNAs and proteins were present. During pregnancy, the expression of Glut 4 decreased progressively, whereas that of Glut 1 increased. In the lactating mammary glan...
متن کاملnano-rods zno as an efficient catalyst for the synthesis of chromene phosphonates, direct amidation and formylation of amines
چکیده ندارد.
Arteriovenous glucose differences across the mammary gland of the fed, starved, and re-fed lactating rat.
Arteriovenous glucose difference across the mammary gland of the lactating rat was used as an 'instantaneous' monitor of mammary glucose uptake. Plasma [glucose] and arteriovenous glucose difference varied according to whether Halothane, diethyl ether or sodium pentobarbitone anaesthesia was used. In pentobarbitone-treated rats a 60% glucose extraction in the fed state decreased to 5% after 18 ...
متن کاملIntracellular Glucose Concentration in Derepressed Yeast Cells Consuming Glucose Is High Enough To Reduce the Glucose
متن کامل
Kinetic mechanism of glucose-6-phosphate dehydrogenase from the lactating rat mammary gland. Implications for regulation.
The steady state kinetic mechanisms for glucose-6-P dehydrogenase from lactating rat mammary glands were derived for the NADP-linked and NAD-linked reactions. Initial velocity studies and inhibition patterns using NADPH and glucosamine-6-P are consistent with sequential mechanisms with random order addition of glucose-6-P and coenzyme. No evidence was found for enzyme isomerization or dead-end ...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Biochemical Journal
سال: 1975
ISSN: 0264-6021
DOI: 10.1042/bj1520153